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Dhakar, K

It supports the development of stage-aware biomarkers of mitochondrial distress and contact-site failure, encourages use of human-relevant and therapeutically timed experimental systems, and identifies selective stabilization of pathological organelle crosstalk as a more realistic goal than global suppression of mitochondrial signaling

Targeting the NAD salvage pathway suppresses APC mutation-driven colorectal cancer growth and Wnt/-catenin signaling via increasing Axin level

doi: 10.1161/STROKEAHA.124.048509

Anal Biochem 666

Monitoring and Long-Term Safety A key advantage of clinic-based peptide therapy is ongoing monitoring